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Interleukin-1β mediated amyloid plaque clearance is independent of CCR2 signaling in the APP/PS1 mouse model of Alzheimer’s disease

Neuroinflammation is a key component of Alzheimer’s disease (AD) pathogenesis. Particularly, the proinflammatory cytokine interleukin-1 beta (IL-1β) is upregulated in human AD and believed to promote amyloid plaque deposition. However, studies from our laboratory have shown that chronic IL-1β overex...

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Detalhes bibliográficos
Main Authors: Rivera-Escalera, Fátima, Matousek, Sarah B., Ghosh, Simantini, Olschowka, John A., O’Banion, M. Kerry
Formato: Artigo
Idioma:Inglês
Publicado em: 2014
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4120473/
https://ncbi.nlm.nih.gov/pubmed/24874542
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.nbd.2014.05.018
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