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Platelets are required for enhanced activation of the endothelium and fibrinogen in a mouse thrombosis model of APS
Antiphospholipid syndrome (APS) is defined by thrombosis, fetal loss, and the presence of antiphospholipid antibodies, including anti–β2-glycoprotein-1 autoantibodies (anti-β2GP1) that have a direct role in the pathogenesis of thrombosis in vivo. The cellular targets of the anti-β2GP1 autoantibody/β...
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| Auteurs principaux: | , , , , |
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| Format: | Artigo |
| Langue: | Inglês |
| Publié: |
American Society of Hematology
2014
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| Sujets: | |
| Accès en ligne: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4110663/ https://ncbi.nlm.nih.gov/pubmed/24825863 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2014-02-554980 |
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