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Site-specific AGE modifications in the ECM: a role for glyoxal in protein damage in diabetes

Non-enzymatic modification of proteins in hyperglycemia is a major proposed mechanism of diabetic complications. Specifically, advanced glycation end products (AGEs) derived from hyperglycemia-induced reactive carbonyl species (RCS) can have pathogenic consequences when they target functionally crit...

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Библиографические подробности
Главные авторы: Voziyan, Paul, Brown, Kyle L., Chetyrkin, Sergei, Hudson, Billy
Формат: Artigo
Язык:Inglês
Опубликовано: 2014
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Online-ссылка:https://ncbi.nlm.nih.gov/pmc/articles/PMC4104777/
https://ncbi.nlm.nih.gov/pubmed/23492568
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1515/cclm-2012-0818
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