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Dimethylfumarate attenuates restenosis after acute vascular injury by cell-specific and Nrf2-dependent mechanisms

Excessive proliferation of vascular smooth muscle cells (VSMCs) and incomplete re-endothelialization is a major clinical problem limiting the long-term efficacy of percutaneous coronary angioplasty. We tested if dimethylfumarate (DMF), an anti-psoriasis drug, could inhibit abnormal vascular remodeli...

Täydet tiedot

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Bibliografiset tiedot
Päätekijät: Oh, Chang Joo, Park, Sungmi, Kim, Joon-Young, Kim, Han-Jong, Jeoung, Nam Ho, Choi, Young-Keun, Go, Younghoon, Park, Keun-Gyu, Lee, In-Kyu
Aineistotyyppi: Artigo
Kieli:Inglês
Julkaistu: Elsevier 2014
Aiheet:
Linkit:https://ncbi.nlm.nih.gov/pmc/articles/PMC4087186/
https://ncbi.nlm.nih.gov/pubmed/25009787
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.redox.2014.06.003
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