Dysfunctional mitochondrial Ca(2+) handling in mutant SOD1 mouse models of fALS: integration of findings from motor neuron somata and motor terminals

Lignende værker

• The Ψ(m) depolarization that accompanies mitochondrial Ca(2+) uptake is greater in mutant SOD1 than in wild-type mouse motor terminals
  af: Nguyen, Khanh T., et al.
  Udgivet: (2009)
• Repetitive Nerve Stimulation Transiently Opens the Mitochondrial Permeability Transition Pore in Motor Nerve Terminals of Symptomatic Mutant SOD1 Mice
  af: Nguyen, Khanh T., et al.
  Udgivet: (2011)
• Stimulation-Evoked Increases in Cytosolic [Ca(2+)] in Mouse Motor Nerve Terminals Are Limited by Mitochondrial Uptake and Are Temperature-Dependent
  af: David, Gavriel, et al.
  Udgivet: (2000)
• Mitochondrial Ca(2+) uptake prevents desynchronization of quantal release and minimizes depletion during repetitive stimulation of mouse motor nerve terminals
  af: David, Gavriel, et al.
  Udgivet: (2003)
• Stimulation-induced mitochondrial [Ca(2+)] elevations in mouse motor terminals: comparison of wild-type with SOD1-G93A
  af: Vila, Lizette, et al.
  Udgivet: (2003)