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Cardiac-specific overexpression of RhoA results in sinus and atrioventricular nodal dysfunction and contractile failure

RhoA is a low-molecular-weight GTPase that has been implicated in the regulation of hypertrophic cardiac muscle cell growth. To study the role of RhoA in control of cardiac function in vivo, transgenic mice expressing wild-type and constitutively activated forms of RhoA under the control of the card...

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Detaylı Bibliyografya
Asıl Yazarlar: Sah, Valerie P., Minamisawa, Susumu, Tam, Steven P., Wu, Thomas H., Dorn, Gerald W., Ross, John, Chien, Kenneth R., Brown, Joan Heller
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: American Society for Clinical Investigation 1999
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC408391/
https://ncbi.nlm.nih.gov/pubmed/10377168
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