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Human Fetal Membranes Generate Distinct Cytokine Profiles in Response to Bacterial Toll-Like Receptor and Nod-Like Receptor Agonists

Bacterial infection-associated inflammation is thought to be a major cause of preterm premature rupture of membranes. Proinflammatory cytokines, such as interleukin 1B (IL1B), can weaken fetal membranes (FM) by upregulating matrix metalloproteinases and inducing apoptosis. The mechanism by which inf...

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書誌詳細
主要な著者: Hoang, Mai, Potter, Julie A., Gysler, Stefan M., Han, Christina S., Guller, Seth, Norwitz, Errol R., Abrahams, Vikki M.
フォーマット: Artigo
言語:Inglês
出版事項: Society for the Study of Reproduction, Inc. 2014
主題:
オンライン・アクセス:https://ncbi.nlm.nih.gov/pmc/articles/PMC4076407/
https://ncbi.nlm.nih.gov/pubmed/24429216
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1095/biolreprod.113.115428
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