Epidermal growth factor receptor (EGFR) and Stat3 signal through Kras and have mutually opposite effects on Cten

Παρόμοια τεκμήρια

• Cten Is Targeted by Kras Signalling to Regulate Cell Motility in the Colon and Pancreas
  ανά: Al-Ghamdi, Saleh, κ.ά.
  Έκδοση: (2011)
• Multiple pathways regulate Cten in colorectal cancer without a Tensin switch
  ανά: Thorpe, Hannah, κ.ά.
  Έκδοση: (2016)
• KRAS and EGFR mutations coexisting in lung adenocarcinoma
  ανά: Sousa, Vitor, κ.ά.
  Έκδοση: (2010)
• CTEN prolongs signaling by EGFR through reducing its ligand-induced degradation
  ανά: Hong, Shiao-Ya, κ.ά.
  Έκδοση: (2013)
• Evidence that synthetic lethality underlies the mutual exclusivity of oncogenic KRAS and EGFR mutations in lung adenocarcinoma
  ανά: Unni, Arun M, κ.ά.
  Έκδοση: (2015)