The effects of MyD88 deficiency on disease phenotype in dysferlin-deficient A/J mice: role of endogenous TLR ligands

An absence of dysferlin leads to activation of innate immune receptors such as Toll-like receptors (TLRs) and skeletal muscle inflammation. Myeloid differentiation primary response gene 88 (MyD88) is a key mediator of TLR-dependent innate immune signalling. We hypothesized that endogenous TLR ligand...

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Main Authors: Uaesoontrachoon, Kitipong, Cha, Hee-Jae, Ampong, Beryl, Sali, Arpana, Vandermeulen, Jack, Wei, Benjamin, Creeden, Brittany, Huynh, Tony, Quinn, James, Tatem, Kathleen, Rayavarapu, Sree, Hoffman, Eric P, Nagaraju, Kanneboyina
Formato: Artigo
Idioma:Inglês
Publicado: 2013
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Article
Acceso en liña:https://ncbi.nlm.nih.gov/pmc/articles/PMC4071457/
https://ncbi.nlm.nih.gov/pubmed/23857504
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/path.4207
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