RIPK1 blocks early postnatal lethality mediated by caspase-8 and RIPK3

Receptor interacting protein kinase (RIPK)-1 is involved in RIPK3-dependent and independent signaling pathways leading to cell death and/or inflammation. Genetic ablation of RIPK1 causes postnatal lethality, which was not prevented by deletion of RIPK3, caspase-8 or FADD. However, animals that lack...

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Main Authors: Dillon, Christopher P., Weinlich, Ricardo, Rodriguez, Diego A., Cripps, James G., Quarato, Giovanni, Gurung, Prajwal, Verbist, Katherine C., Brewer, Taylor L., Llambi, Fabien, Gong, Yi-Nan, Janke, Laura J., Kelliher, Michelle A., Kanneganti, Thirumala-Devi, Green, Douglas R.
פורמט: Artigo
שפה:Inglês
יצא לאור: 2014
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Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC4068710/
https://ncbi.nlm.nih.gov/pubmed/24813850
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.cell.2014.04.018
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