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Reversing the Deleterious Effects of α2-antiplasmin on Tissue Plasminogen Activator Therapy Improves Outcomes in Experimental Ischemic Stroke
High blood levels of α2-antiplasmin have been associated with failed tissue plasminogen activator (TPA) therapy for ischemic stroke. Yet, other data suggests that α2-antiplasmin may be protective in stroke, because it defends against bleeding and excitotoxicity. To address this paradox, we examined...
Gorde:
| Egile Nagusiak: | , , |
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| Formatua: | Artigo |
| Hizkuntza: | Inglês |
| Argitaratua: |
2014
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| Gaiak: | |
| Sarrera elektronikoa: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4066326/ https://ncbi.nlm.nih.gov/pubmed/24556477 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2014.02.009 |
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