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Endothelial injury due to eNOS deficiency accelerates the progression of chronic renal disease in the mouse
The vascular endothelium expresses endothelial nitric oxide synthase (eNOS) that generates nitric oxide (NO) to help maintain vascular integrity due to its anti-inflammatory, antiproliferative, and antithrombogenic effects. Pharmacological blockade of NO production has been shown to exacerbate renal...
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| Autors principals: | , , , , , , , , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
American Physiological Society
2009
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4063508/ https://ncbi.nlm.nih.gov/pubmed/19036847 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1152/ajprenal.90450.2008 |
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