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Impaired repair of cyclobutane pyrimidine dimers in human keratinocytes deficient in p53 and p63
While many p53-deficient cell types are impaired in global genomic nucleotide excision repair of cyclobutane pyrimidine dimers (CPDs), human epidermal keratinocytes expressing human papillomavirus type 16 E6 and E7 are p53 deficient and yet maintain repair of CPD. We hypothesized that the p53 homolo...
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| Main Authors: | , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2007
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4061977/ https://ncbi.nlm.nih.gov/pubmed/17984111 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1093/carcin/bgm244 |
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