Retinal ganglion cell (RGC) programmed necrosis contributes to ischemia-reperfusion-induced retinal damage

Lignende værker

• Toll-like receptor 4 contributes to retinal ischemia/reperfusion injury
  af: Dvoriantchikova, Galina, et al.
  Udgivet: (2010)
• Tumor necrosis factor-alpha mediates activation of NF-κB and JNK signaling cascades in retinal ganglion cells and astrocytes in opposite ways
  af: Dvoriantchikova, Galina, et al.
  Udgivet: (2014)
• Neuronal NAD(P)H Oxidases Contribute to ROS Production and Mediate RGC Death after Ischemia
  af: Dvoriantchikova, Galina, et al.
  Udgivet: (2012)
• The effect of extrinsic Wnt/β-catenin signaling in Muller glia on retinal ganglion cell (RGC) neurite growth
  af: Musada, Ganeswara Rao, et al.
  Udgivet: (2020)
• The TIR-domain-containing adapter inducing interferon-β-dependent signaling cascade plays a crucial role in ischemia–reperfusion-induced retinal injury, whereas the contribution of the myeloid differentiation primary response 88-dependent signaling cascade is not as pivotal
  af: Dvoriantchikova, Galina, et al.
  Udgivet: (2014)