RAGE Gene Deletion Inhibits the Development and Progression of Ductal Neoplasia and Prolongs Survival in a Murine Model of Pancreatic Cancer

BACKGROUND: The receptor for advanced glycation end-products (RAGE) is implicated in pancreatic tumorigenesis. Activating Kras mutations and p16 inactivation are genetic abnormalities most commonly detected as pancreatic ductal epithelium progresses from intraepithelial neoplasia (PanIN) to adenocar...

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Detaylı Bibliyografya
Asıl Yazarlar: DiNorcia, Joseph, Lee, Minna K., Moroziewicz, Dorota N., Winner, Megan, Suman, Paritosh, Bao, Fei, Remotti, Helen E., Zou, Yu Shan, Yan, Shi Fang, Qiu, Wanglong, Su, Gloria H., Schmidt, Ann Marie, Allendorf, John D.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 2011
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Article
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC4049447/
https://ncbi.nlm.nih.gov/pubmed/22052106
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11605-011-1754-9
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