RAGE Gene Deletion Inhibits the Development and Progression of Ductal Neoplasia and Prolongs Survival in a Murine Model of Pancreatic Cancer

BACKGROUND: The receptor for advanced glycation end-products (RAGE) is implicated in pancreatic tumorigenesis. Activating Kras mutations and p16 inactivation are genetic abnormalities most commonly detected as pancreatic ductal epithelium progresses from intraepithelial neoplasia (PanIN) to adenocar...

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Detalhes bibliográficos
Main Authors: DiNorcia, Joseph, Lee, Minna K., Moroziewicz, Dorota N., Winner, Megan, Suman, Paritosh, Bao, Fei, Remotti, Helen E., Zou, Yu Shan, Yan, Shi Fang, Qiu, Wanglong, Su, Gloria H., Schmidt, Ann Marie, Allendorf, John D.
Formato: Artigo
Idioma:Inglês
Publicado em: 2011
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Article
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC4049447/
https://ncbi.nlm.nih.gov/pubmed/22052106
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1007/s11605-011-1754-9
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