HIV-1 STIMULATES NUCLEAR ENTRY OF AMYLOID BETA VIA DYNAMIN DEPENDENT EEA1 AND TGF-β/SMAD SIGNALING

Clinical evidence indicates increased amyloid deposition in HIV-1-infected brains, which contributes to neurocognitive dysfunction in infected patients. Here we show that HIV-1 exposure stimulates amyloid beta (Aβ) nuclear entry in human brain endothelial cells (HBMEC), the main component of the blo...

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Main Authors: András, Ibolya E., Toborek, Michal
Format: Artigo
Sprog:Inglês
Udgivet: 2014
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Article
Online adgang:https://ncbi.nlm.nih.gov/pmc/articles/PMC4019515/
https://ncbi.nlm.nih.gov/pubmed/24491918
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2014.01.027
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