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HIV-1 STIMULATES NUCLEAR ENTRY OF AMYLOID BETA VIA DYNAMIN DEPENDENT EEA1 AND TGF-β/SMAD SIGNALING
Clinical evidence indicates increased amyloid deposition in HIV-1-infected brains, which contributes to neurocognitive dysfunction in infected patients. Here we show that HIV-1 exposure stimulates amyloid beta (Aβ) nuclear entry in human brain endothelial cells (HBMEC), the main component of the blo...
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| Autors principals: | , |
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| Format: | Artigo |
| Idioma: | Inglês |
| Publicat: |
2014
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| Matèries: | |
| Accés en línia: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4019515/ https://ncbi.nlm.nih.gov/pubmed/24491918 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yexcr.2014.01.027 |
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