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CaMKII Binding to GluN2B Is Differentially Affected by Macromolecular Crowding Reagents
Binding of the Ca(2+)/calmodulin(CaM)-dependent protein kinase II (CaMKII) to the NMDA-type glutamate receptor (NMDAR) subunit GluN2B controls long-term potentiation (LTP), a form of synaptic plasticity thought to underlie learning and memory. Regulation of this interaction is well-studied biochemic...
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| Main Authors: | , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
Public Library of Science
2014
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC4010494/ https://ncbi.nlm.nih.gov/pubmed/24796865 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0096522 |
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