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Enzymatic Activity of CaMKII Is Not Required for Its Interaction with the Glutamate Receptor Subunit GluN2B

Binding of the Ca(2+)/calmodulin (CaM)-dependent protein kinase II (CaMKII) to the NMDA-type glutamate receptor subunit GluN2B is an important control mechanism for the regulation of synaptic strength. CaMKII binding to GluN2B and CaMKII translocation to synapses are induced by an initial Ca(2+)/CaM...

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Detalhes bibliográficos
Main Authors: Barcomb, Kelsey, Coultrap, Steven J., Bayer, K. Ulrich
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Pharmacology and Experimental Therapeutics 2013
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3834147/
https://ncbi.nlm.nih.gov/pubmed/24056996
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1124/mol.113.089045
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