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Molecular Basis for Increased Risk for Late-onset Alzheimer Disease Due to the Naturally Occurring L28P Mutation in Apolipoprotein E4

The apolipoprotein (apo) E4 isoform has consistently emerged as a susceptibility factor for late-onset Alzheimer disease (AD), although the exact mechanism is not clear. A rare apoE4 mutant, apoE4[L28P] Pittsburgh, burdens carriers with an added risk for late-onset AD and may be a useful tool for ga...

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Autori principali: Argyri, Letta, Dafnis, Ioannis, Theodossiou, Theodossis A., Gantz, Donald, Stratikos, Efstratios, Chroni, Angeliki
Natura: Artigo
Lingua:Inglês
Pubblicazione: American Society for Biochemistry and Molecular Biology 2014
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Accesso online:https://ncbi.nlm.nih.gov/pmc/articles/PMC4007480/
https://ncbi.nlm.nih.gov/pubmed/24644280
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1074/jbc.M113.538124
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