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PolyQ Disease: Too Many Q’s, Too Much Function?

The nature of the gain-of-function toxicity found in polyglutamine (polyQ) diseases has been the subject of considerable debate. Duvick et al. (2010) and Nedelsky et al. (2010) now show that in two of these diseases, pathology is mediated by normal protein activity.

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Podrobná bibliografie
Hlavní autoři: Kratter, Ian H., Finkbeiner, Steven
Médium: Artigo
Jazyk:Inglês
Vydáno: 2010
Témata:
On-line přístup:https://ncbi.nlm.nih.gov/pmc/articles/PMC3989162/
https://ncbi.nlm.nih.gov/pubmed/20869586
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.neuron.2010.09.012
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