Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice

BACKGROUND: Traumatic brain injury (TBI) enhances pro-inflammatory responses, neuronal loss and long-term behavioral deficits. Caveolins (Cavs) are regulators of neuronal and glial survival signaling. Previously we showed that astrocyte and microglial activation is increased in Cav-1 knock-out (KO)...

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Autors principals: Niesman, Ingrid R, Schilling, Jan M, Shapiro, Lee A, Kellerhals, Sarah E, Bonds, Jacqueline A, Kleschevnikov, Alexander M, Cui, Weihua, Voong, April, Krajewski, Stan, Ali, Sameh S, Roth, David M, Patel, Hemal H, Patel, Piyush M, Head, Brian P
Format: Artigo
Idioma:Inglês
Publicat: BioMed Central 2014
Matèries:
Research
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3975903/
https://ncbi.nlm.nih.gov/pubmed/24593993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-11-39
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