Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice

BACKGROUND: Traumatic brain injury (TBI) enhances pro-inflammatory responses, neuronal loss and long-term behavioral deficits. Caveolins (Cavs) are regulators of neuronal and glial survival signaling. Previously we showed that astrocyte and microglial activation is increased in Cav-1 knock-out (KO)...

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Detalhes bibliográficos
Main Authors: Niesman, Ingrid R, Schilling, Jan M, Shapiro, Lee A, Kellerhals, Sarah E, Bonds, Jacqueline A, Kleschevnikov, Alexander M, Cui, Weihua, Voong, April, Krajewski, Stan, Ali, Sameh S, Roth, David M, Patel, Hemal H, Patel, Piyush M, Head, Brian P
Formato: Artigo
Idioma:Inglês
Publicado em: BioMed Central 2014
Assuntos:
Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3975903/
https://ncbi.nlm.nih.gov/pubmed/24593993
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1186/1742-2094-11-39
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