Overexpression of miR-125a in Myelodysplastic Syndrome CD34(+) Cells Modulates NF-κB Activation and Enhances Erythroid Differentiation Arrest

Lignende værker

• Overexpression of miR-125a in myelodysplastic syndrome CD34+ cells modulates NF-κB activation and enhances erythroid differentiation arrest.
  af: Irene Gañán-Gómez, et al.
  Udgivet: (2014-01-01)
• Global H3K4me3 genome mapping reveals alterations of innate immunity signaling and overexpression of JMJD3 in human myelodysplastic syndrome CD34+ cells
  af: Wei, Yue, et al.
  Udgivet: (2013)
• Overexpression of the Toll-Like Receptor (TLR) Signaling Adaptor MYD88, but Lack of Genetic Mutation, in Myelodysplastic Syndromes
  af: Dimicoli, Sophie, et al.
  Udgivet: (2013)
• Reactive oxygen species levels control NF-κB activation by low dose deferasirox in erythroid progenitors of low risk myelodysplastic syndromes
  af: Meunier, Mathieu, et al.
  Udgivet: (2017)
• Implications of discrepancy in morphologic diagnosis of myelodysplastic syndrome between referral and tertiary care centers
  af: Naqvi, Kiran, et al.
  Udgivet: (2011)