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Calpain-mediated proteolysis of tropomodulin isoforms leads to thin filament elongation in dystrophic skeletal muscle

Duchenne muscular dystrophy (DMD) induces sarcolemmal mechanical instability and rupture, hyperactivity of intracellular calpains, and proteolytic breakdown of muscle structural proteins. Here we identify the two sarcomeric tropomodulin (Tmod) isoforms, Tmod1 and Tmod4, as novel proteolytic targets...

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Detalhes bibliográficos
Main Authors: Gokhin, David S., Tierney, Matthew T., Sui, Zhenhua, Sacco, Alessandra, Fowler, Velia M.
Formato: Artigo
Idioma:Inglês
Publicado em: The American Society for Cell Biology 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3952854/
https://ncbi.nlm.nih.gov/pubmed/24430868
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1091/mbc.E13-10-0608
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