The MEK/ERK Pathway is the Primary Conduit for Borrelia burgdorferi-Induced Inflammation and P53-Mediated Apoptosis in Oligodendrocytes

Lignende værker

• Non-viable Borrelia burgdorferi induce inflammatory mediators and apoptosis in human oligodendrocytes
  af: Parthasarathy, Geetha, et al.
  Udgivet: (2013)
• Intracellular TLR7 is activated in human oligodendrocytes in response to Borrelia burgdorferi exposure
  af: Parthasarathy, Geetha, et al.
  Udgivet: (2018)
• Receptor tyrosine kinases play a significant role in human oligodendrocyte inflammation and cell death associated with the Lyme disease bacterium Borrelia burgdorferi
  af: Parthasarathy, Geetha, et al.
  Udgivet: (2017)
• A possible role for inflammation in mediating apoptosis of oligodendrocytes as induced by the Lyme disease spirochete Borrelia burgdorferi
  af: Ramesh, Geeta, et al.
  Udgivet: (2012)
• Inflammatory mediator release from primary rhesus microglia in response to Borrelia burgdorferi results from the activation of several receptors and pathways
  af: Parthasarathy, Geetha, et al.
  Udgivet: (2015)