The Mos/MAP kinase pathway stabilizes c-Fos by phosphorylation and augments its transforming activity in NIH 3T3 cells.

Podobne knjige/članki

• pp39mos is associated with p34cdc2 kinase in c-mosxe-transformed NIH 3T3 cells.
  od: Zhou, R, et al.
  Izdano: (1992)
• Alteration of a cyclic AMP-dependent protein kinase phosphorylation site in the c-Fos protein augments its transforming potential.
  od: Tratner, I, et al.
  Izdano: (1992)
• Transformation‐specific Decrease of Phosphorylation of 80K Protein, a Substrate of Protein Kinase C, in NIH3T3 Cells
  od: Oh‐uchida, Mamoru, et al.
  Izdano: (1990)
• Abrogation of translation initiation factor eIF-2 phosphorylation causes malignant transformation of NIH 3T3 cells.
  od: Donzé, O, et al.
  Izdano: (1995)
• Correlation between Physiological and Transforming Activities of the c–mos Proto–oncogene Product and Identification of an Essential Mos Domain for These Activities
  od: Okazaki, Kenji, et al.
  Izdano: (1991)