Antisense Oligonucleotide Against GSK-3β in Brain of SAMP8 Mice Improves Learning and Memory and Decreases Oxidative Stress: Involvement of Transcription Factor Nrf2 and Implications for Alzheimer Disease

Lignende værker

• Antisense directed against PS-1 gene decreases brain oxidative markers in aged senescence accelerated mice (SAMP8) and reverses learning and memory impairment: a proteomics study
  af: Fiorini, Ada, et al.
  Udgivet: (2013)
• Central and Peripheral Administration of Antisense Oligonucleotide Targeting Amyloid Precursor Protein Improves Learning and Memory and Reduces Neuroinflammatory Cytokines in Tg2576 (APPswe) Mice
  af: Farr, Susan A., et al.
  Udgivet: (2014)
• Decreased Levels of PSD95 and Two Associated Proteins and Increased Levels of BCl(2) and Caspase 3 in Hippocampus from Subjects with Amnestic Mild Cognitive Impairment: Insights into Their Potential Roles for Loss of Synapses and Memory, Accumulation of Aβ, and Neurodegeneration in a Prodromal Stage of Alzheimer's Disease
  af: Sultana, Rukhsana, et al.
  Udgivet: (2010)
• Delivery of Antisense Oligonucleotides to the Cornea
  af: Chau, Viet Q., et al.
  Udgivet: (2020)
• Oxidatively modified, mitochondria-relevant brain proteins in subjects with Alzheimer disease and mild cognitive impairment
  af: Sultana, Rukhsana, et al.
  Udgivet: (2009)