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JNK promotes Bax translocation to mitochondria through phosphorylation of 14-3-3 proteins
Targeted gene disruption studies have established that the c-Jun NH(2)-terminal kinase (JNK) is required for the stress-induced release of mitochondrial cytochrome c and apoptosis, and that the Bax subfamily of Bcl-2-related proteins is essential for JNK-dependent apoptosis. However, the mechanism b...
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| Autores principales: | , , , , , , , , |
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| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
Nature Publishing Group
2004
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.gov/pmc/articles/PMC394248/ https://ncbi.nlm.nih.gov/pubmed/15071501 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/sj.emboj.7600194 |
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