Autosomal locus regulates inverse relationship between sialic acid content and capacity of mouse erythrocytes to activate human alternative complement pathway

Ítems similars

• Regulation by membrane sialic acid of β1H-dependent decay-dissociation of amplification C3 convertase of the alternative complement pathway
  per: Fearon, Douglas T.
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  per: Fearon, DT, et al.
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• An autosomal dominant gene regulates the extent of 9-O-acetylation of murine erythrocyte sialic acids. A probable explanation for the variation in capacity to activate the human alternate complement pathway
  Publicat: (1980)
• Properdin: initiation of alternative complement pathway.
  per: Fearon, D T, et al.
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• Regulation of the amplification C3 convertase of human complement by an inhibitory protein isolated from human erythrocyte membrane
  per: Fearon, Douglas T.
  Publicat: (1979)