Autosomal locus regulates inverse relationship between sialic acid content and capacity of mouse erythrocytes to activate human alternative complement pathway

Documenti analoghi

• Regulation by membrane sialic acid of β1H-dependent decay-dissociation of amplification C3 convertase of the alternative complement pathway
  di: Fearon, Douglas T.
  Pubblicazione: (1978)
• Activation of the alternative complement pathway with rabbit erythrocytes by circumvention of the regulatory action of endogenous control proteins
  di: Fearon, DT, et al.
  Pubblicazione: (1977)
• An autosomal dominant gene regulates the extent of 9-O-acetylation of murine erythrocyte sialic acids. A probable explanation for the variation in capacity to activate the human alternate complement pathway
  Pubblicazione: (1980)
• Properdin: initiation of alternative complement pathway.
  di: Fearon, D T, et al.
  Pubblicazione: (1975)
• Regulation of the amplification C3 convertase of human complement by an inhibitory protein isolated from human erythrocyte membrane
  di: Fearon, Douglas T.
  Pubblicazione: (1979)