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Combination therapy in a transgenic model of Alzheimer’s disease
The pathological accumulation of the β-amyloid protein (Aβ) has been closely associated with synaptic loss and neurotoxicity contributing to cognitive dysfunction in Alzheimer’s disease (AD). Oligomers of Aβ42 appear to be the most neurotoxic form. Two of the most promising attempts to reduce Aβ acc...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3926203/ https://ncbi.nlm.nih.gov/pubmed/24120437 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.expneurol.2013.10.001 |
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