Complement C3 convertase: Cell surface restriction of β1H control and generation of restriction on neuraminidase-treated cells
The alternative or properdin pathway of complement is primarily controlled by the endopeptidase C3b inactivator (C3bINA) and the nonproteolytic glycoprotein β1H. The molecular mechanisms of control were investigated by performing binding studies of radiolabeled complement proteins to C3b bearing she...
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| Publicado en: | Proc Natl Acad Sci U S A |
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| Autores principales: | , |
| Formato: | Artigo |
| Lenguaje: | Inglês |
| Publicado: |
National Academy of Sciences
1978
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| Materias: | |
| Acceso en línea: | https://ncbi.nlm.nih.govhttps://pmc.ncbi.nlm.nih.gov/articles/PMC392564/ https://ncbi.nlm.nih.govhttps://pubmed.ncbi.nlm.nih.gov/276881/ https://ncbi.nlm.nih.govhttps://doi.org/10.1073/pnas.75.5.2416 |
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