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Complement C3 convertase: Cell surface restriction of β1H control and generation of restriction on neuraminidase-treated cells

The alternative or properdin pathway of complement is primarily controlled by the endopeptidase C3b inactivator (C3bINA) and the nonproteolytic glycoprotein β1H. The molecular mechanisms of control were investigated by performing binding studies of radiolabeled complement proteins to C3b bearing she...

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Detaylı Bibliyografya
Asıl Yazarlar: Pangburn, Michael K., Müller-Eberhard, Hans J.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: 1978
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC392564/
https://ncbi.nlm.nih.gov/pubmed/276881
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