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Angiotensin II stimulates cardiac fibroblast migration via the differential regulation of matrixins and RECK

Sustained induction and activation of matrixins (matrix metalloproteinases or MMPs), and the destruction and deposition of extracellular matrix (ECM), are the hallmarks of cardiac fibrosis. The reversion-inducing-cysteine-rich protein with Kazal motifs (RECK) is a unique membrane-anchored endogenous...

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Detalhes bibliográficos
Main Authors: Siddesha, Jalahalli M., Valente, Anthony J., Sakamuri, Siva S.V.P., Yoshida, Tadashi, Gardner, Jason D., Somanna, Naveen, Takahashi, Chiaki, Noda, Makoto, Chandrasekar, Bysani
Formato: Artigo
Idioma:Inglês
Publicado em: 2013
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Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3896127/
https://ncbi.nlm.nih.gov/pubmed/24095877
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.yjmcc.2013.09.015
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