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Carbon Monoxide Confers Protection in Sepsis by Enhancing Beclin 1-Dependent Autophagy and Phagocytosis

Aims: Sepsis, a systemic inflammatory response to infection, represents the leading cause of death in critically ill patients. However, the pathogenesis of sepsis remains incompletely understood. Carbon monoxide (CO), when administered at low physiologic doses, can modulate cell proliferation, apopt...

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Detaylı Bibliyografya
Asıl Yazarlar: Lee, Seonmin, Lee, Seon-Jin, Coronata, Anna A., Fredenburgh, Laura E., Chung, Su Wol, Perrella, Mark A., Nakahira, Kiichi, Ryter, Stefan W., Choi, Augustine M.K.
Materyal Türü: Artigo
Dil:Inglês
Baskı/Yayın Bilgisi: Mary Ann Liebert, Inc. 2014
Konular:
Online Erişim:https://ncbi.nlm.nih.gov/pmc/articles/PMC3894711/
https://ncbi.nlm.nih.gov/pubmed/23971531
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2013.5368
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