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Carbon Monoxide Confers Protection in Sepsis by Enhancing Beclin 1-Dependent Autophagy and Phagocytosis

Aims: Sepsis, a systemic inflammatory response to infection, represents the leading cause of death in critically ill patients. However, the pathogenesis of sepsis remains incompletely understood. Carbon monoxide (CO), when administered at low physiologic doses, can modulate cell proliferation, apopt...

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Autors principals: Lee, Seonmin, Lee, Seon-Jin, Coronata, Anna A., Fredenburgh, Laura E., Chung, Su Wol, Perrella, Mark A., Nakahira, Kiichi, Ryter, Stefan W., Choi, Augustine M.K.
Format: Artigo
Idioma:Inglês
Publicat: Mary Ann Liebert, Inc. 2014
Matèries:
Accés en línia:https://ncbi.nlm.nih.gov/pmc/articles/PMC3894711/
https://ncbi.nlm.nih.gov/pubmed/23971531
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2013.5368
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