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Carbon Monoxide Confers Protection in Sepsis by Enhancing Beclin 1-Dependent Autophagy and Phagocytosis

Aims: Sepsis, a systemic inflammatory response to infection, represents the leading cause of death in critically ill patients. However, the pathogenesis of sepsis remains incompletely understood. Carbon monoxide (CO), when administered at low physiologic doses, can modulate cell proliferation, apopt...

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Detalhes bibliográficos
Main Authors: Lee, Seonmin, Lee, Seon-Jin, Coronata, Anna A., Fredenburgh, Laura E., Chung, Su Wol, Perrella, Mark A., Nakahira, Kiichi, Ryter, Stefan W., Choi, Augustine M.K.
Formato: Artigo
Idioma:Inglês
Publicado em: Mary Ann Liebert, Inc. 2014
Assuntos:
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3894711/
https://ncbi.nlm.nih.gov/pubmed/23971531
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1089/ars.2013.5368
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