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IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4(+) T cells but not CD8(+) T cells(1)

IFN-γ is generally believed to be important in the autoimmune pathogenesis of type 1 diabetes (T1D). However, the development of spontaneous β cell autoimmunity is unaffected in NOD mice lacking expression of IFN-γ or the IFN-γ receptor (IFNγR), bringing into question the role IFN-γ has in T1D. In t...

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Bibliografske podrobnosti
Main Authors: Yi, Zuoan, Li, Li, Garland, Alaina, He, Qiuming, Wang, Haidong, Katz, Jonathan D, Tisch, Roland, Wang, Bo
Format: Artigo
Jezik:Inglês
Izdano: 2012
Teme:
Online dostop:https://ncbi.nlm.nih.gov/pmc/articles/PMC3883988/
https://ncbi.nlm.nih.gov/pubmed/22865049
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.201242374
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