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IFN-γ receptor deficiency prevents diabetes induction by diabetogenic CD4(+) T cells but not CD8(+) T cells(1)
IFN-γ is generally believed to be important in the autoimmune pathogenesis of type 1 diabetes (T1D). However, the development of spontaneous β cell autoimmunity is unaffected in NOD mice lacking expression of IFN-γ or the IFN-γ receptor (IFNγR), bringing into question the role IFN-γ has in T1D. In t...
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| Main Authors: | , , , , , , , |
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| Format: | Artigo |
| Jezik: | Inglês |
| Izdano: |
2012
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| Teme: | |
| Online dostop: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3883988/ https://ncbi.nlm.nih.gov/pubmed/22865049 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1002/eji.201242374 |
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