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Virally-expressed connexin26 restores gap junction function in the cochlea of conditional Gjb2 knockout mice
Mutations in GJB2, which codes for the gap junction protein connexin26, are the most common causes of human nonsyndromic hereditary deafness. We inoculated modified adeno-associated viral vectors into the scala media of early postnatal conditional Gjb2 knockout mice to drive exogenous connexin26 exp...
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| 主要な著者: | , , , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2013
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| 主題: | |
| オンライン･アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3881370/ https://ncbi.nlm.nih.gov/pubmed/24225640 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1038/gt.2013.59 |
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