Hepatitis C Virus NS3/4A Protease Inhibits Complement Activation by Cleaving Complement Component 4

BACKGROUND: It has been hypothesized that persistent hepatitis C virus (HCV) infection is mediated in part by viral proteins that abrogate the host immune response, including the complement system, but the precise mechanisms are not well understood. We investigated whether HCV proteins are involved...

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Main Authors: Mawatari, Seiichi, Uto, Hirofumi, Ido, Akio, Nakashima, Kenji, Suzuki, Tetsuro, Kanmura, Shuji, Kumagai, Kotaro, Oda, Kohei, Tabu, Kazuaki, Tamai, Tsutomu, Moriuchi, Akihiro, Oketani, Makoto, Shimada, Yuko, Sudoh, Masayuki, Shoji, Ikuo, Tsubouchi, Hirohito
פורמט: Artigo
שפה:Inglês
יצא לאור: Public Library of Science 2013
נושאים:
Research Article
גישה מקוונת:https://ncbi.nlm.nih.gov/pmc/articles/PMC3861371/
https://ncbi.nlm.nih.gov/pubmed/24349192
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1371/journal.pone.0082094
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