eNOS-derived nitric oxide regulates endothelial barrier function through VE-cadherin and Rho GTPases

Transient disruption of endothelial adherens junctions and cytoskeletal remodeling are responsible for increases in vascular permeability induced by inflammatory stimuli and vascular endothelial growth factor (VEGF). Nitric oxide (NO) produced by endothelial NO synthase (eNOS) is crucial for VEGF-in...

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Bibliographische Detailangaben
Hauptverfasser: Di Lorenzo, Annarita, Lin, Michelle I., Murata, Takahisa, Landskroner-Eiger, Shira, Schleicher, Michael, Kothiya, Milankumar, Iwakiri, Yasuko, Yu, Jun, Huang, Paul L., Sessa, William C.
Format: Artigo
Sprache:Inglês
Veröffentlicht: The Company of Biologists 2013
Schlagworte:
Research Article
Online Zugang:https://ncbi.nlm.nih.gov/pmc/articles/PMC3860306/
https://ncbi.nlm.nih.gov/pubmed/24046447
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1242/jcs.115972
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