Acute Inhibition of Excessive Mitochondrial Fission After Myocardial Infarction Prevents Long‐term Cardiac Dysfunction

BACKGROUND: Ischemia and reperfusion (IR) injury remains a major cause of morbidity and mortality and multiple molecular and cellular pathways have been implicated in this injury. We determined whether acute inhibition of excessive mitochondrial fission at the onset of reperfusion improves mitochond...

Disgrifiad llawn

Wedi'i Gadw mewn:
Manylion Llyfryddiaeth
Prif Awduron: Disatnik, Marie‐Hélène, Ferreira, Julio C.B., Campos, Juliane Cruz, Gomes, Kátia Sampaio, Dourado, Paulo M.M., Qi, Xin, Mochly‐Rosen, Daria
Fformat: Artigo
Iaith:Inglês
Cyhoeddwyd: Blackwell Publishing Ltd 2013
Pynciau:
Original Research
Mynediad Ar-lein:https://ncbi.nlm.nih.gov/pmc/articles/PMC3835263/
https://ncbi.nlm.nih.gov/pubmed/24103571
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.113.000461
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