Acute Inhibition of Excessive Mitochondrial Fission After Myocardial Infarction Prevents Long‐term Cardiac Dysfunction

BACKGROUND: Ischemia and reperfusion (IR) injury remains a major cause of morbidity and mortality and multiple molecular and cellular pathways have been implicated in this injury. We determined whether acute inhibition of excessive mitochondrial fission at the onset of reperfusion improves mitochond...

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Autores principales: Disatnik, Marie‐Hélène, Ferreira, Julio C.B., Campos, Juliane Cruz, Gomes, Kátia Sampaio, Dourado, Paulo M.M., Qi, Xin, Mochly‐Rosen, Daria
Formato: Artigo
Lenguaje:Inglês
Publicado: Blackwell Publishing Ltd 2013
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Original Research
Acceso en línea:https://ncbi.nlm.nih.gov/pmc/articles/PMC3835263/
https://ncbi.nlm.nih.gov/pubmed/24103571
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.113.000461
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