Acute Inhibition of Excessive Mitochondrial Fission After Myocardial Infarction Prevents Long‐term Cardiac Dysfunction

BACKGROUND: Ischemia and reperfusion (IR) injury remains a major cause of morbidity and mortality and multiple molecular and cellular pathways have been implicated in this injury. We determined whether acute inhibition of excessive mitochondrial fission at the onset of reperfusion improves mitochond...

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Detalhes bibliográficos
Main Authors: Disatnik, Marie‐Hélène, Ferreira, Julio C.B., Campos, Juliane Cruz, Gomes, Kátia Sampaio, Dourado, Paulo M.M., Qi, Xin, Mochly‐Rosen, Daria
Formato: Artigo
Idioma:Inglês
Publicado em: Blackwell Publishing Ltd 2013
Assuntos:
Original Research
Acesso em linha:https://ncbi.nlm.nih.gov/pmc/articles/PMC3835263/
https://ncbi.nlm.nih.gov/pubmed/24103571
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1161/JAHA.113.000461
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