Experimental sepsis-induced mitochondrial biogenesis is dependent on autophagy, TLR4, and TLR9 signaling in liver

Organ injury in sepsis is initially characterized by dysfunction without cell death and structural damage, and thus with the ability to recover organ function. Adaptive metabolic responses to sepsis can prevent bioenergetic failure and death. These studies were aimed at investigating the influence o...

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Bibliographic Details
Main Authors: Carchman, Evie H., Whelan, Sean, Loughran, Patricia, Mollen, Kevin, Stratamirovic, Sladjana, Shiva, Sruti, Rosengart, Matthew R., Zuckerbraun, Brian S.
Format: Artigo
Language:Inglês
Published: Federation of American Societies for Experimental Biology 2013
Subjects:
Research Communications
Online Access:https://ncbi.nlm.nih.gov/pmc/articles/PMC3834775/
https://ncbi.nlm.nih.gov/pubmed/23982147
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1096/fj.13-229476
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