Amyloid beta-induced Glycogen Synthase Kinase 3β Phosphorylated VDAC1 in Alzheimer’s Disease: Implications for Synaptic Dysfunction and Neuronal Damage

Lignende værker

• Abnormal interaction of VDAC1 with amyloid beta and phosphorylated tau causes mitochondrial dysfunction in Alzheimer's disease
  af: Manczak, Maria, et al.
  Udgivet: (2012)
• Amyloid beta, mitochondrial dysfunction and synaptic damage: implications for cognitive decline in aging and Alzheimer’s disease
  af: Reddy, P. Hemachandra, et al.
  Udgivet: (2008)
• Abnormal interaction of oligomeric amyloid-β with phosphorylated tau: implications to synaptic dysfunction and neuronal damage
  af: Manczak, Maria, et al.
  Udgivet: (2013)
• Aging and Amyloid beta-induced oxidative DNA damage and mitochondrial dysfunction in Alzheimer's Disease: Implications for early intervention and therapeutics
  af: Mao, Peizhong, et al.
  Udgivet: (2011)
• Activation of glycogen synthase kinase-3 beta mediates β-amyloid induced neuritic damage in Alzheimer's disease(,,)
  af: DaRocha-Souto, B., et al.
  Udgivet: (2011)