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Lipopolysaccharide stimulates platelets through an IL-1β autocrine loop
Lipopolysaccharide (LPS) activates platelets through TLR4, aiding productive sepsis, with stimulated splicing and translation of stored heteronuclear pro-Interleukin-1β (IL-1β)RNA. Although the IL1R1 receptor for IL1 shares downstream components with the TLR4 receptor, platelets are not known to exp...
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| 主要な著者: | , , , , |
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| フォーマット: | Artigo |
| 言語: | Inglês |
| 出版事項: |
2013
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| 主題: | |
| オンライン・アクセス: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3818355/ https://ncbi.nlm.nih.gov/pubmed/24081990 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.4049/jimmunol.1300354 |
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