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Overexpression of SCYL1-BP1 stabilizes functional p53 by suppressing MDM2-mediated ubiquitination

Previously, we defined SCYL1-BP1 to be a substrate of Pirh2 that binds to MDM2. In the current study, we found that an increase in SCYL1-BP1 protein levels caused a parallel change in the amount of p53 protein due to the inhibition by SCYL-BP1 of MDM2-mediated p53 ubiquitination. SCYL1-BP1 was not a...

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Détails bibliographiques
Auteurs principaux: Yan, Jing, Di, Yujun, Shi, Huili, Rao, Hai, Huo, Keke
Format: Artigo
Langue:Inglês
Publié: 2010
Sujets:
Accès en ligne:https://ncbi.nlm.nih.gov/pmc/articles/PMC3789512/
https://ncbi.nlm.nih.gov/pubmed/20849854
https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1016/j.febslet.2010.09.019
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