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ATF3 Protects against LPS-Induced Inflammation in Mice via Inhibiting HMGB1 Expression
Lipopolysaccharide (LPS) triggers innate immunity mainly via TLR4 signaling. ATF3 is a negative regulator of TLR4 signaling. HMGB1 plays a critical role in the final step of sepsis. However, the mechanisms of ATF3 and the role of HMGB1 in regulating innate immunity-induced sepsis are incompletely un...
שמור ב:
| Main Authors: | , , , , , |
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| פורמט: | Artigo |
| שפה: | Inglês |
| יצא לאור: |
Hindawi Publishing Corporation
2013
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| נושאים: | |
| גישה מקוונת: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3770047/ https://ncbi.nlm.nih.gov/pubmed/24062788 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1155/2013/716481 |
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