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The β2 integrin–kindlin-3 interaction is essential for T-cell homing but dispensable for T-cell activation in vivo
Kindlin-3 is mutated in the rare genetic disorder, leukocyte adhesion deficiency type III, which is characterized by deficient integrin-mediated adhesion of leukocytes and platelets. However, the specific roles of kindlin-3–β2-integrin interactions in T-cell adhesion and homing and immune responses...
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| Main Authors: | , , , , , |
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| Formato: | Artigo |
| Idioma: | Inglês |
| Publicado em: |
American Society of Hematology
2013
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| Assuntos: | |
| Acesso em linha: | https://ncbi.nlm.nih.gov/pmc/articles/PMC3750339/ https://ncbi.nlm.nih.gov/pubmed/23823319 https://ncbi.nlm.nih.govhttp://dx.doi.org/10.1182/blood-2013-02-484998 |
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